Steroids carry a risk that many patients, and even some prescribing doctors, overlook. They can silently raise the pressure inside your eye. And raised eye pressure, left unchecked, damages the optic nerve and causes glaucoma, says Dr Shibal Bhartiya. Timely monitoring, not waiting for symptoms, is what prevents irreversible optic nerve damage.

Steroids are powerful medicines. Doctors use them to treat inflammation, autoimmune disease, allergies, and dozens of other conditions. But they can trigger a silent rise in eye pressure, often without early symptoms.

This condition is called steroid-induced glaucoma. It is one of the most preventable causes of serious vision loss in India.

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Dr Shibal Bhartiya is a fellowship-trained glaucoma specialist and Mayo Clinic Research Collaborator with over 25 years of experience. Her approach focuses on identifying risk before damage is irreversible, simplifying treatment decisions, and protecting vision long-term. Emphasis on early detection, risk assessment, and continuity of care. She is rated 5 stars across 1,500+ patient reviews on Google.

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What Are Steroids and Why Do Doctors Use Them?

Steroids, specifically corticosteroids, reduce inflammation in the body. Doctors prescribe them in many forms: eye drops, oral tablets, inhalers, nasal sprays, skin creams, and injections directly into or around the eye.

Common brand names include prednisolone, dexamethasone, betamethasone, triamcinolone, and budesonide. Many are available over the counter in India without a prescription. This is a serious problem.

People often self-medicate with steroid eye drops for redness or allergy, sometimes for months, without any eye pressure monitoring.

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How Do Steroids Raise Eye Pressure?

Your eye constantly produces a fluid called aqueous humour. This fluid drains out through a mesh-like structure called the trabecular meshwork. Steroids interfere with this drainage. The fluid builds up. Pressure inside the eye rises.

This process is called a steroid response. It does not happen to everyone. But certain people are far more susceptible. Glaucoma patients, first-degree relatives of glaucoma patients, people with high myopia, and diabetics have a higher risk of becoming steroid responders.

In a steroid responder, eye pressure can rise significantly, sometimes within days of starting treatment. More often, the rise is gradual and goes unnoticed for weeks or months.

The danger is that raised eye pressure causes no pain. No redness. No blurring. You feel nothing until the optic nerve is already damaged.

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Which Steroids Carry the Highest Risk?

Eye drops carry the greatest risk. They deliver steroids directly into the eye in concentrated form. Potent drops like prednisolone and dexamethasone raise eye pressure more than weaker formulations like fluorometholone or loteprednol. Duration matters too: the longer the use, the greater the risk.

Periocular injections, injections around the eye used in uveitis and retinal disease, release steroids slowly over weeks to months. Triamcinolone acetonide injections are a particularly common cause of prolonged eye pressure elevation. Once the depot is in place, it cannot be removed easily.

Oral steroids carry a lower but real risk, especially with prolonged use at high doses.

Inhaled steroids for asthma and COPD, and nasal sprays for allergic rhinitis, carry a small but measurable risk, particularly with long-term use.

Skin creams applied around the eyes can absorb through the eyelid skin and raise eye pressure. This is underappreciated and often missed.

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Symptoms of Steroid-Induced Glaucoma

In most cases, there are no symptoms. This is what makes steroid-induced glaucoma dangerous.

By the time vision changes become noticeable, significant optic nerve damage has often already occurred. Peripheral vision goes first — and most people do not notice peripheral vision loss until it is severe.

In rare cases, when eye pressure rises very rapidly, patients may experience headache, eye ache, blurring, or haloes around lights. But this is the exception, not the rule.

The only way to detect steroid-induced glaucoma early is to check eye pressure regularly while on any steroid therapy, especially eye drops.

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How Is Steroid-Induced Glaucoma Diagnosed?

Diagnosis requires a full glaucoma evaluation. This includes:

Tonometry measures eye pressure. Normal pressure is usually between 10 and 21 mmHg. Steroid responders may reach 30, 40, or even higher.

Gonioscopy examines the drainage angle to confirm the trabecular meshwork is open, as it is in steroid glaucoma, distinguishing it from angle-closure glaucoma.

OCT (Optical Coherence Tomography) scans the optic nerve and the nerve fibre layer to detect structural damage before vision loss is symptomatic.

Visual field testing maps the field of vision to detect functional loss.

Optic disc examination allows direct visualisation of the nerve head for signs of damage and cupping.

Steroid-induced glaucoma looks identical to primary open-angle glaucoma on examination. The distinguishing clue is the history: elevated pressure that developed after starting a steroid, and that improves when the steroid is stopped or changed.

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Is Steroid-Induced Glaucoma Reversible?

The short answer: sometimes, if caught early enough.

In many patients, stopping or switching the steroid allows eye pressure to normalise within weeks. If the optic nerve has not been damaged, the condition is fully reversible.

But optic nerve damage is permanent. Glaucoma does not recover. If pressure has been high long enough to injure the nerve, even partially, that damage remains even after the steroid is stopped.

This is why early detection is critical. A short course of steroid eye drops that goes unmonitored can cause permanent vision loss that no treatment can reverse.

Caught early, steroid glaucoma is one of the most manageable forms of glaucoma. That is why monitoring matters.

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Treatment Options

Step one is always to reconsider the steroid.

Can the dose be reduced? Can the steroid be stopped? Is there a possibility of using a less potent formulation? For eye drops, switching from prednisolone to fluorometholone or loteprednol often reduces the pressure response significantly.

Sometimes the underlying condition, uveitis, for example, requires continued steroid treatment. In these cases, eye pressure must be managed medically.

Pressure-lowering eye drops are the first line of treatment. The same drops used in primary glaucoma: prostaglandin analogues, beta-blockers, carbonic anhydrase inhibitors, and alpha agonists, are effective in steroid glaucoma.

Laser treatment (SLT) can improve drainage through the trabecular meshwork and reduce dependence on drops.

Surgery: trabeculectomy or a glaucoma drainage device , is reserved for cases where drops and laser do not control pressure adequately. Surgery in steroid glaucoma is generally highly effective.

For patients who have received a periocular steroid injection and cannot have it removed, sustained medical treatment is the mainstay until the depot is absorbed.

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The Indian Context: A Hidden Epidemic

India has a particular problem with steroid-induced glaucoma. Steroid eye drops are widely available without prescription. Patients self-treat for red eyes, allergy, and post-operative care, often on the advice of pharmacists or non-specialist practitioners.

Many patients arrive in my clinic having used potent steroid drops every day for six, twelve, or even twenty-four months. Their pressure is grossly elevated. The optic nerves are damaged. Their peripheral vision is affected and will not return.

This is preventable. Every patient using steroid eye drops needs their eye pressure monitored. Every patient on long-term systemic steroids deserves at least an annual eye check. This is not optional.

As a fellowship-trained glaucoma specialist seeing patients from across India, Dr Bhartiya offers structured steroid glaucoma risk assessments for patients on long-term steroid therapy, including those referred by other treating doctors.

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When Should You See a Glaucoma Specialist?

See a fellowship-trained glaucoma specialist if:

• You are using steroid eye drops for more than two weeks
• You have been prescribed a periocular steroid injection
• You are on long-term oral steroids and have never had your eye pressure checked
• You have a family history of glaucoma and are on any steroid therapy
• You are a known glaucoma patient who requires steroids for any reason
• Your eye pressure has been noted to be high on a routine eye check
• If you have been told your eye pressure is high while on steroids, an independent glaucoma second opinion can clarify whether treatment or monitoring is needed.

Do not wait for symptoms. There are none, until it is too late. Bring your steroid prescription and any previous eye pressure readings to your appointment.

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Clinical Reality (What’s not always obvious)

• Steroid-induced glaucoma is often silent in the early stages
• Vision may remain completely normal on routine testing
• Pressure rise can happen within weeks in some patients, but months in others
• Not all steroids are equal — eye drops, skin creams, inhalers, and even nasal sprays can contribute
• The response is individual — some people are “steroid responders” without knowing it
• Stopping the steroid does not always reverse the damage completely
• Damage, once established, follows the same irreversible course as primary glaucoma

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What Actually Helps (And What Doesn’t)

What helps:

• Early identification of steroid use (even non-ocular forms)
• Baseline and follow-up intraocular pressure monitoring
• Switching to safer alternatives where possible
• Timely initiation of anti-glaucoma therapy if needed
• Long-term monitoring even after stopping steroids

What doesn’t help:

• Assuming “short-term use is always safe”
• Ignoring non-eye steroid sources (dermatology creams, inhalers)
• Relying only on vision clarity as a marker of safety
• Delaying evaluation because symptoms are absent
• Repeated steroid prescriptions without pressure monitoring

Remember This

Situation / Trigger	What Patients Often Assume	Clinical Reality	What Should Be Done
Using steroid eye drops	“Doctor prescribed it, so it’s safe”	Even prescribed steroids can raise eye pressure	Monitor IOP within weeks of starting
Using skin creams near eyes	“It’s just topical, not affecting eyes”	Periocular absorption can increase eye pressure	Inform ophthalmologist and monitor
Using inhalers for asthma	“It doesn’t reach the eye”	Chronic use can contribute to pressure rise	Periodic eye pressure checks
Short-term steroid use	“Too brief to cause harm”	Some individuals respond rapidly	Early follow-up is essential
No symptoms	“If I see well, everything is fine”	Glaucoma damage is silent initially	Regular screening, not symptom-based
Stopping steroids	“Problem is solved now”	Damage may persist or progress	Continued monitoring required
Multiple steroid prescriptions	“Different doctors, different issues”	Cumulative exposure increases risk	Centralised tracking of steroid use

Frequently Asked Questions

Can steroid eye drops cause glaucoma even when used for a short time?

A brief course, less than two weeks, rarely causes a clinically significant pressure rise. But risk increases with duration and potency. Any steroid eye drop use lasting more than two weeks warrants a pressure check.

How long does it take for steroids to raise eye pressure?

In highly susceptible individuals, pressure can rise within days. In most steroid responders, the rise occurs over two to six weeks of use. With depot injections, pressure may continue to rise for months.

Does stopping the steroid cure steroid glaucoma?

It normalises the pressure in most patients, yes. But if the optic nerve has already been damaged, that damage is permanent. Stopping the steroid does not restore lost vision.

Can inhaled steroids for asthma cause glaucoma?

Yes, though the risk is lower than with eye drops. Long-term use of high-dose inhaled corticosteroids has been associated with a modest increase in glaucoma risk, particularly in patients who already have elevated eye pressure.

Can steroid skin creams cause glaucoma?

Yes. Creams applied to the face and eyelid skin can absorb into the eye in meaningful amounts. This is an underrecognised cause of steroid-induced ocular hypertension.

What is a steroid responder?

A steroid responder is someone whose eye pressure rises significantly on steroid therapy. Roughly 5% of the general population are high responders. Glaucoma patients, first-degree relatives of glaucoma patients, high myopes, and diabetics have a much higher rate of response.

Is steroid glaucoma the same as regular glaucoma?

The optic nerve damage is identical. The mechanism of pressure elevation differs: steroids impair drainage through the trabecular meshwork. The treatment approach is similar, but the critical first step is always to reassess and if possible stop or reduce the causative steroid.

Can I still use steroids if I have glaucoma?

Yes, but only under close specialist supervision with frequent pressure monitoring. Never use steroid eye drops without the oversight of an ophthalmologist if you have a diagnosis of glaucoma or a family history of the condition.

I had a steroid injection around my eye six months ago and my pressure is still high. What should I do?

This is a recognised complication of periocular depot steroids. The injection releases slowly over months. Pressure management with drops or laser is usually required until the depot is absorbed. See a glaucoma specialist, this situation requires careful, ongoing monitoring.

What should I do if my pharmacist gives me steroid eye drops for a red eye?

Do not use steroid eye drops without a diagnosis from an ophthalmologist. Red eyes have many causes, viral conjunctivitis, allergic conjunctivitis, dry eye, most of which do not always require steroids and some of which can be worsened by them. Always get a proper diagnosis before using any steroid eye drop.

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Book a consultation with Dr Shibal Bhartiya:

Marengo Asia Hospitals, Gurugram

Phone: +91 88826 38735

Website: drshibalbhartiya.com

Google Business Profile: maps.app.goo.gl/mcfegmHTuhqV5hSp6

Read the research articles

This article was written by Dr Shibal Bhartiya, fellowship-trained glaucoma specialist and Mayo Clinic Research Collaborator, Clinical Director at Marengo Asia Hospitals, Gurugram, known for ethical, patient-centred glaucoma care and independent glaucoma second opinions. This article was edited in April 2026.

She has published peer-reviewed research on glaucoma management, examining how treatment decisions should balance medical evidence, patient preferences, and long-term vision outcomes.

As Editor-in-Chief of Clinical and Experimental Vision and Eye Research and Executive Editor of the Journal of Current Glaucoma Practice (Pubmed Indexed, official journal of the International Society of Glaucoma Surgery), Dr Shibal Bhartiya brings editorial and research depth to every clinical decision. Her 200+ publications, including 90+ PubMed-indexed publications and 28 edited textbooks span glaucoma biology, surgical outcomes, health equity, and emerging diagnostics.

Available on Pubmed and Google Scholar

Dr Shibal Bhartiya
Glaucoma • Second Opinion • Advanced Care

www.drshibalbhartiya.com
 +91 88826 38735

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