A Routine Eye Check and Glaucoma

A routine eye check may raise suspicion of glaucoma through elevated eye pressure, changes in the optic nerve, or unexplained vision changes. However, confirming glaucoma usually requires specialised tests such as optic nerve imaging, visual field testing, and corneal thickness measurement. Also, a routine for glasses is not a substitute for an eye exam, as the former can often miss glaucoma.

Glaucoma usually causes no pain, no redness, and no obvious vision change in its early stages. Most people with glaucoma feel completely normal until significant and irreversible damage has occurred. The only way to detect it early is a comprehensive eye examination that includes optic nerve assessment, intraocular pressure measurement, and visual field testing

A Routine Eye Check Revealed a Sight-Threatening Disease

Mrs SG came to see me because her glasses prescription had not felt right for a few months. She was 57. She worked at a desk. Her eyes were tired by evening, and she assumed she needed a stronger number. She had no pain. No redness. No alarming moment that made her think something was wrong.

Her previous optician had given her a new prescription six months earlier. It had not helped. She booked an appointment with me because a colleague had suggested a second opinion.

I examined her in the usual way. Her visual acuity was reasonable. Her anterior segment was quiet. Then I checked her retina and optic nerve.

The optic nerve in her left eye had a cup that was too large. The rim tissue was thinning at the inferior pole. Her intraocular pressure was 24 mmHg in the right eye and 26 in the left. I asked her to sit with the visual field machine.

The field test confirmed what the disc had suggested. There was a dense arcuate defect in her left eye. A significant portion of her peripheral vision was already gone. She had not noticed. You rarely do with glaucoma, because the brain fills in the gaps until it cannot.

She did not need a stronger glasses prescription. She had glaucoma, and it had been quietly advancing for what was likely several years.

Patient details have been changed to protect privacy.


Remember

Sunita’s case is not unusual. Glaucoma is called the silent thief of sight for a reason. It causes no pain, no visible redness, and no early warning that most patients would recognise. By the time vision loss is noticeable, the disease has already caused permanent damage. In India, an estimated 12 million people have glaucoma, and almost 90% of them do not know it. (The Chennai glaucoma Study).

Below, I explain what glaucoma actually does to the eye, why it is so reliably missed, and which symptoms, or absences of symptoms, should prompt an urgent examination.


What Glaucoma Actually Does to Your Eye

Glaucoma is a disease of the optic nerve. The optic nerve carries visual information from the eye to the brain. When this nerve is damaged, that information is lost permanently. No treatment can restore what is already gone. Treatment can only slow or stop further damage.

In most cases, the damage is caused or worsened by raised pressure inside the eye. This pressure, called intraocular pressure or IOP, builds when fluid inside the eye does not drain properly. The drainage system becomes less efficient over time, pressure rises, and the optic nerve fibres begin to die. The process is painless in the vast majority of patients.

What makes glaucoma particularly deceptive is the pattern of vision loss. It begins at the periphery, the edges of your visual field. The brain compensates automatically. Both eyes together create a complete picture, and each eye covers for the blind spots of the other. Patients often do not notice peripheral vision loss until more than 40 percent of their optic nerve fibres have already been destroyed. By that point, the disease is well advanced.

In SG’s case, her glasses prescription had changed slightly because her visual system was compensating for early field loss. It was not a refractive change. It was her brain working harder to make sense of incomplete information. This pattern, subtle visual dissatisfaction without a clear cause, is one of the most common presentations I see in patients who turn out to have early to moderate glaucoma.


Glaucoma vs Normal Ageing: How to Tell the Difference

Symptom or SignWhat It SuggestsWhat To Do
Gradual blurring that a new glasses prescription does not fixMay indicate optic nerve or macular pathology, not refractive changeSee an ophthalmologist for optic nerve assessment, not just a refraction
Difficulty adjusting from bright to dim lightCan be an early sign of peripheral field lossRequest a visual field test at your next eye appointment
Frequent glasses changes with no lasting improvementSuggests the problem is not the prescriptionAsk for intraocular pressure measurement and disc evaluation
Mild headache or eye heaviness without rednessIn some patients, mildly elevated IOP causes subtle discomfortCheck IOP, especially if over 40 or with family history of glaucoma
No symptoms at all, but a family member has glaucomaFirst-degree relatives have a 4 to 9 times higher riskSchedule a comprehensive glaucoma screening even if you feel completely well
Squinting or tilting the head to see clearlyMay indicate undetected visual field asymmetryFull field test for both eyes separately

Why Glaucoma Is So Often Missed

The most common reason glaucoma goes undetected is that a routine glasses check is not a glaucoma examination.

When a patient visits an optician or a basic eye clinic for a new prescription, the standard assessment measures visual acuity and refraction. It does not always include optic nerve photography, intraocular pressure measurement, or visual field testing. These are the three investigations that detect glaucoma. Without all three, the disease is invisible.

Sunita had seen an optician twice in three years. Her visual acuity was checked each time. Her optic nerve was never examined.

The second reason glaucoma is missed is the absence of symptoms. Patients present to doctors when something feels wrong. Glaucoma does not feel wrong, not for years. There is no cultural expectation in India of an annual comprehensive eye examination. Most people attend only when they need a new prescription or when something is visibly red or painful. By those criteria, a glaucoma patient has no reason to come at all.

The third reason is that IOP alone is not a reliable screening tool. Many patients with glaucoma have pressure in the so-called normal range. Normal-tension glaucoma accounts for a substantial proportion of cases, particularly in people of Asian descent. A single IOP reading of 16 mmHg does not exclude the diagnosis.

SG’s IOP was elevated, which made diagnosis more straightforward. But many of my patients with confirmed glaucoma have had pressures that would not have triggered concern at a routine check.


When To See an Eye Specialist

See an ophthalmologist for a comprehensive glaucoma assessment if any of the following apply:

  • A parent, sibling, or child has been diagnosed with glaucoma
  • You are over 40 and have not had a comprehensive eye examination in the past two years
  • You have been told your eye pressure is “a little high” but were not referred further
  • You have changed your glasses prescription twice in two years with no lasting improvement
  • You have diabetes, as this increases glaucoma risk
  • You are of South Asian, East Asian, or African descent, all of which carry higher glaucoma risk
  • You use steroid eye drops, nasal sprays, or inhalers long-term
  • You were told everything was fine, but your vision still does not feel right

A comprehensive assessment takes around 30 to 45 minutes and is painless. It will include optic nerve imaging, IOP measurement, corneal thickness assessment, and a visual field test. This combination reliably detects glaucoma at a stage when treatment can prevent significant vision loss.


Frequently Asked Questions

Can you have glaucoma with normal eye pressure?

Yes. Normal-tension glaucoma is a recognised and common form of the disease, particularly in people of Asian descent. A normal IOP reading does not rule out glaucoma; optic nerve assessment and visual field testing are essential.

Does glaucoma always cause pain?

No. The most common forms of glaucoma are completely painless. Pain is associated with acute angle-closure glaucoma, which is a sudden and rare presentation. Most patients with chronic open-angle glaucoma, the most prevalent type, feel nothing at all until vision loss is advanced.

Can lost vision from glaucoma be restored?

No. Optic nerve damage caused by glaucoma is permanent. Treatment with eye drops, laser, or surgery can slow or stop further damage, but vision already lost cannot be recovered. Early detection is the only way to protect useful sight.

How often should I have a glaucoma check if I have a family history?

If a first-degree relative has glaucoma, you should have a comprehensive eye examination every year from the age of 40, or earlier if your ophthalmologist advises it.


Book a Consultation

If you have a family history of glaucoma, have not had a comprehensive eye examination in the past two years, or have been told your eye pressure is elevated, a dedicated assessment is worth arranging now. The earlier glaucoma is found, the more vision can be protected.

At Dr Shibal Bhartiya Eye Clinic, Gurugram, a glaucoma assessment includes optic nerve imaging, visual field testing, corneal thickness measurement, and a full review of your risk profile. [second opinion]

[Book an Appointment → +91 88826 38735]


This page is a part of the Glaucoma Hub. you may want to read about Glaucoma Progression, and Risk Stratification in Glaucoma. Other articles of interest could be Advanced Glaucoma Care in Gurgaon, What Good Glaucoma Care Actually Optimises For, What Happens If Glaucoma Is Left Untreated?, More Glaucoma Eye Drops is Not Better Glaucoma Care, 5 Mistakes Patients Make in Glaucoma Care and Do You Really Need Treatment for Glaucoma?


About the Author

This article was written by Dr Shibal Bhartiya, fellowship-trained glaucoma specialist and Mayo Clinic Research Collaborator, Clinical Director at Marengo Asia Hospitals, Gurugram, known for ethical, patient-centred glaucoma care and independent glaucoma second opinions. She is also the Program Director for Community Outreach & Wellness; and for the Marengo Asia International Institute of Neuro and Spine.

She has published peer-reviewed research on glaucoma management, examining how treatment decisions should balance medical evidence, patient preferences, and long-term vision outcomes.

As Editor-in-Chief of Clinical and Experimental Vision and Eye Research and Executive Editor of the Journal of Current Glaucoma Practice (Pubmed Indexed, official journal of the International Society of Glaucoma Surgery), Dr Shibal Bhartiya brings editorial and research depth to every clinical decision. Her 200+ publications, including 90+ PubMed-indexed publications and 28 edited textbooks span glaucoma biology, surgical outcomes, health equity, and emerging diagnostics.

1500+ Five Star Patient Reviews Google Business Profile

If you are unable to come to Dr Bhartiya’s clinic: Read more about teleconsultation

Read her research on PubMed | Google Scholar | ResearchGate | ORCID

Upload your reports for a structured review.| www.drshibalbhartiya.com | +91 88826 38735

Leave a review on Google

Why Do I Need Glaucoma Treatment If My Vision Seems Normal?

Glaucoma often causes permanent optic nerve damage long before noticeable vision loss develops. Treatment is designed to protect your future vision by slowing or preventing progression before symptoms appear, Dr Shibal Bhartiya explains.

Your vision feels fine. No pain, no blur, no obvious change. So why is your doctor urging treatment? This is the most common question glaucoma patients ask, and it deserves a direct, honest answer,

Glaucoma destroys your optic nerve silently. By the time you notice something is wrong, you have already lost nerve fibres that will never return. Treatment does not restore what is gone. It protects what remains.


The Vision You Have Now Is Not the Vision You Started With

Glaucoma removes peripheral vision first. Your central vision stays sharp until the disease is advanced. Your brain also compensates, filling in blind areas so skilfully that you do not notice them. You may have lost 30 to 40 percent of your optic nerve fibres before any symptom appears.

This is why “I can see fine” is not a safe reassurance in glaucoma. It reflects the vision that has survived, not the vision that has been lost.


Why Glaucoma Treatment Feels Unnecessary (And Why That Feeling Is Dangerous)

Glaucoma drops do not improve your vision. They do not reduce pain because glaucoma causes none. They do not change how things look today. Their only job is to lower the pressure inside your eye and slow the damage to your optic nerve.

When a treatment produces no felt benefit, stopping it feels harmless. This is the central psychological trap in glaucoma care. Patients who feel well skip doses, delay refills, or discontinue treatment altogether. The nerve continues to deteriorate. By the time symptoms appear, the loss is severe and permanent.

The absence of symptoms is not evidence that you are safe. It is evidence that the disease has not yet crossed your threshold of awareness.


What the Research Actually Shows

Studies consistently show that controlling eye pressure reduces the risk of glaucoma progression. The Ocular Hypertension Treatment Study showed that lowering pressure by 20 percent reduced conversion to glaucoma by more than half. The Early Manifest Glaucoma Trial showed that each mmHg reduction in pressure produced a measurable reduction in progression risk.

You are not treating a feeling. You are treating a measurable biological risk that happens to produce no warning before it causes irreversible harm.


“But My Pressures Are Controlled Now — Do I Still Need Drops?”

Yes. Controlled pressure means the treatment is working. Stopping treatment removes the protection. Pressure typically rises again within days to weeks after discontinuation.

Some patients assume that normal pressure readings mean the problem is resolved. Glaucoma is a chronic condition. Controlled pressure is a maintained state, not a cured one.


Normal-Tension Glaucoma: When Pressure Is Not Even the Full Story

A significant group of patients develop glaucoma with eye pressures in the statistically normal range. Their optic nerves are still vulnerable, often due to poor blood flow, structural susceptibility, or other factors. For these patients, the question “but my pressure is fine” does not mean treatment is unnecessary. It means the target pressure needs to be set lower, and other risk factors need attention.

This is one reason that glaucoma management requires individual assessment, not a one-size guideline.


FAQ

If I have no symptoms, does that mean my glaucoma is mild?

Not necessarily. Glaucoma can cause significant optic nerve damage before any symptom appears. The severity of glaucoma is assessed through structural tests like OCT and functional tests like visual fields, not through how your vision feels day to day.

What happens if I skip my glaucoma drops for a few days?

Eye pressure can rise within 24 to 48 hours of stopping treatment. Over time, this pressure exposure adds to cumulative nerve damage. Occasional missed doses are less harmful than long gaps, but no dose-skipping is risk-free in active glaucoma.

Can I know if my glaucoma is getting worse?

Progression is detected through serial OCT scans and visual field testing, not through symptoms. This is why regular follow-up is essential even when your vision feels unchanged.

My doctor wants to change my drops. Should I get a second opinion first?

A second opinion is always appropriate in glaucoma, especially if you are uncertain about treatment changes, surgical recommendations, or whether your current regimen is adequate. Glaucoma causes irreversible loss, so the cost of a wrong decision is permanent.

Are there people who do not need treatment despite a glaucoma diagnosis?

In very early suspected glaucoma or ocular hypertension with low risk factors, observation may be appropriate rather than immediate treatment. This is a clinical judgement based on your individual risk profile, your optic nerve appearance, and your visual field results. It requires an experienced glaucoma specialist to make that call correctly.


What You Should Expect From Your Glaucoma Care

A good glaucoma consultation does more than prescribe drops. It establishes your target pressure based on your stage of disease, your age, and your life expectancy. Also, it identifies your progression rate through serial testing. It reviews whether your current treatment is achieving that target. And it explains, clearly, what is at stake if treatment is inconsistent.

If you have left a consultation without understanding why your specific pressure target was chosen, that is worth asking about. If you are uncertain whether your glaucoma is stable or progressing, that is worth investigating through formal visual field and OCT trend analysis.


A Note on Seeking a Second Opinion

Glaucoma decisions carry permanent consequences. Second opinions are not a sign of distrust toward your current doctor. They are a rational response to a disease where the cost of under-treatment is irreversible. An independent review of your scans and pressure history can confirm that you are on the right path, or catch something that has been missed.


This page is part of the Glaucoma Hub hub. Read about our full approach to glaucoma care. Please also read our Second Opinion Hub. Please also read Glaucoma Diagnosis, first 90 days; and Glaucoma Treatment

Here’s another heartening patient story: Tired of drops


About the Author

This article was written by Dr Shibal Bhartiya, fellowship-trained glaucoma specialist and Mayo Clinic Research Collaborator, Clinical Director at Marengo Asia Hospitals, Gurugram, known for ethical, patient-centred glaucoma care and independent glaucoma second opinions. She is also the Program Director for Community Outreach & Wellness; and for the Marengo Asia International Institute of Neuro and Spine.

She has published peer-reviewed research on glaucoma management, examining how treatment decisions should balance medical evidence, patient preferences, and long-term vision outcomes.

As Editor-in-Chief of Clinical and Experimental Vision and Eye Research and Executive Editor of the Journal of Current Glaucoma Practice (Pubmed Indexed, official journal of the International Society of Glaucoma Surgery), Dr Shibal Bhartiya brings editorial and research depth to every clinical decision. Her 200+ publications, including 90+ PubMed-indexed publications and 28 edited textbooks span glaucoma biology, surgical outcomes, health equity, and emerging diagnostics.

1500+ Five Star Patient Reviews Google Business Profile

If you are unable to come to Dr Bhartiya’s clinic: Read more about teleconsultation

Read her research on PubMed | Google Scholar | ResearchGate | ORCID

Upload your reports for a structured review.| www.drshibalbhartiya.com | +91 88826 38735

Leave a review on Google


Glaucoma in India: Why the Risk Is Higher Than You Think

Glaucoma is the most common cause of irreversible blindness in India, and 90% of cases remain undiagnosed. That means nine out of every ten people with glaucoma in this country do not know they have it. An estimated 11.2 million Indians aged 40 and above have glaucoma. And angle closure glaucoma is more common in India, than in the West, says Dr Shibal Bhartiya. Glaucoma in India is often missed or undertreated because it progresses silently, even when vision and eye pressure appear normal. Good glaucoma care focuses on early detection, risk-based monitoring, and long-term protection of vision, not just adding more eye drops.

Glaucoma does not give you a warning. You lose peripheral vision first. By the time you notice something is wrong, damage is already done. The good news is that glaucoma detected early is highly manageable. Blindness from glaucoma is largely preventable with timely diagnosis and consistent treatment.

In India, this story plays out every day at a scale that is hard to comprehend. Dr Shibal Bhartiya, fellowship trained glaucoma specialist in Gurgaon, explains more about Glaucoma in India, and Indians.

Dr Shibal Bhartiya is a fellowship-trained glaucoma specialist and Mayo Clinic Research Collaborator with over 25 years of experience. Her approach focuses on identifying risk before damage is irreversible, simplifying treatment decisions, and protecting vision long-term. Emphasis on early detection, risk assessment, and continuity of care. She is rated 5 stars across 1,500+ patient reviews on Google.


Why Indians Are at Higher Risk

Indians carry specific anatomical and genetic traits that raise their glaucoma risk. One of the most significant is a shallower anterior chamber angle. This makes angle-closure glaucoma far more common in Indian and South Asian eyes than in Western populations.

Primary angle-closure disease may affect as many as 27.6 million Indians. Patients with primary angle-closure glaucoma are twice as likely to go blind compared to those with open-angle glaucoma. Yet this form is frequently missed or misdiagnosed.

Indians also present with glaucoma at a younger age than patients in Western populations. Juvenile open-angle glaucoma, which begins between the ages of 16 and 40, is well documented in Indian tertiary centre data. A family history of glaucoma raises your personal risk significantly. If a parent or sibling has glaucoma, your chances of developing it are much higher.

Other risk factors specific to the Indian context include:

Steroid use without medical supervision, myopia (short-sightedness), diabetes, and a family history of glaucoma are all important risk factors to discuss with your doctor.


The Problem of Late Presentation

Most glaucoma in India is detected late. Very late.

In India, undetected and untreated glaucoma leads to faster progression, earlier visual impairment, and preventable blindness. The core reason is that glaucoma causes no pain and no blur in the early stages. People feel completely fine. They see no reason to visit an eye doctor.

By the time central vision is affected, up to 90% of peripheral nerve fibres may already be lost. That damage cannot be reversed. No surgery, no medication, and no intervention brings that vision back.

This is what makes early screening so critical. You cannot feel glaucoma coming. You can only catch it on examination.


The Scale of the Problem in India

Glaucoma prevalence among Indians aged 40 and above ranges between 2.7% and 4.3% across multiple population-based studies. In those over 70 years of age, the risk rises sharply. Studies show glaucoma affects over 8% of Indians in their seventies and over 14% of those above 80.

Glaucoma is a leading cause of irreversible blindness globally, and the burden in Asia and India is expected to grow substantially by 2040.

India does not have enough glaucoma specialists to manage this burden. Most patients are diagnosed and managed by general ophthalmologists. Structured, specialist-led care makes a real difference to outcomes.


What Makes Glaucoma in Indians Different to Manage

Treating glaucoma in an Indian patient requires a different approach than using a standard Western protocol.

Indian eyes tend to have thinner corneas. Corneal thickness affects how accurately we measure intraocular pressure (IOP). A thin cornea can make the pressure appear lower than it actually is. This leads to underdiagnosis and undertreatment. Also, thinner corneas are an independent risk factor for glaucoma progression.

Angle-closure disease needs gonioscopy, a specialised examination to assess the drainage angle of the eye. Studies have found that a significant proportion of patients in India are incorrectly treated for open-angle glaucoma when they actually have angle-closure disease.The treatment for these two types is fundamentally different.

Normal tension glaucoma (NTG), where optic nerve damage occurs despite normal eye pressure, is also seen in Indian patients. This form requires looking beyond IOP and addressing other risk factors including blood pressuresleep patterns, and vascular health.


How I Approach Glaucoma in Indian Patients

I have spent 25+ years specialising in glaucoma. I see this disease in its full Indian context, not through a textbook written for another population.

My clinical approach includes a full angle assessment with gonioscopy for every new patient, corneal thickness measurement to ensure accurate pressure readings, structural imaging with OCT to detect early nerve fibre loss, visual field analysis (with special emphasis on reliability criteria) and a detailed risk factor review including family history, steroid use, and systemic health.

Correct classification, open-angle versus angle-closure, changes treatment completely. Getting this right at the first visit prevents years of inadequate care.

I also believe in clear communication. Glaucoma is a lifelong condition. You need to understand what you have, why treatment matters, and what to monitor. I take the time to explain this at every visit.

If you have a family history of glaucoma, are over 40, have diabetes, are short-sighted, or use steroid eye drops, you need a glaucoma screening now.


Clinical Reality (What’s Not Always Obvious in Glaucoma Care in India)

  • Normal vision does not mean no glaucoma
    Many patients read 6/6 and still have significant optic nerve damage.
  • Symptoms are often absent until late
    Glaucoma is typically silent — by the time patients notice vision loss, it is often irreversible.
  • Eye pressure (IOP) alone is not enough
    Patients can progress despite “normal” pressures — especially in normal-tension glaucoma, which is common in India.
  • Tests in isolation can mislead
    A single OCT or visual field report cannot define disease. Progression over time is what matters.
  • Cataract and glaucoma often coexist — but are not interchangeable explanations
    Improving vision after cataract surgery does not mean glaucoma risk is gone.
  • More medications ≠ better control
    Multiple drops without a clear long-term plan often reflect escalation without strategy.
  • Follow-up gaps are a major cause of vision loss
    Irregular monitoring is one of the biggest real-world failures in glaucoma care.
  • Family history is under-recognised and under-screened
    Many high-risk individuals in India are never examined until damage has already occurred.

What Good Glaucoma Care Looks Like (Indian Context)

  • Early risk identification — even before symptoms
    Screening is guided by age, family history, corneal thickness, optic nerve appearance — not just complaints.
  • Baseline documentation and longitudinal tracking
    OCT and visual fields are used to establish a baseline and detect change, not just diagnose once.
  • Target pressure is individualised
    Treatment is tailored based on stage of disease, risk profile, and rate of progression — not a fixed number.
  • Medication strategy is structured, not reactive
    Each drop has a purpose. Escalation is thoughtful, not additive.
  • Patient understanding is prioritised
    Patients are told what to watch for: subtle visual changes, adherence issues, side effects.
  • Consistency over intensity
    Regular follow-up (every 3–6 months depending on risk) matters more than aggressive but irregular care.
  • Second opinions are used appropriately
    Especially when:
    • Disease is progressing despite treatment
    • Multiple medications are being used
    • Surgery is being considered
  • The goal is not just seeing clearly — but seeing safely for life
    Glaucoma care is long-term risk management, not short-term vision correction.

Remember

SituationWhat Patients Often AssumeClinical Reality (India Context)What Good Care Looks Like
Vision is normal“I can see clearly, so everything is fine”Glaucoma can cause optic nerve damage even with 6/6 visionRisk-based screening and optic nerve evaluation, even without symptoms
No symptoms“No discomfort means no disease”Glaucoma is silent until late stagesEarly detection through structured exams, not symptom-driven visits
Eye pressure (IOP)“My pressure is normal, so I’m safe”Progression can occur even at normal IOP (common in India)Individualised target IOP based on risk and progression
Single test reports“My OCT/field test is normal”One report is not enough — change over time mattersBaseline + serial comparison to detect progression
Cataract vs glaucoma“Cataract surgery fixed my vision, so I’m okay”Cataract improvement can mask underlying glaucomaParallel evaluation of optic nerve even in cataract patients
Multiple eye drops“More drops means stronger treatment”Overmedication may reflect lack of strategyStructured medication plan with defined goals
Follow-up gaps“I’ll come back if I feel a problem”Irregular follow-up is a major cause of preventable vision lossScheduled monitoring every 3–6 months based on risk
Family history“No one told me to get checked”High-risk individuals often remain unscreened in IndiaProactive screening for family members
Treatment approach“Doctor will adjust if needed”Reactive care often misses slow progressionLong-term planning with defined targets and timelines
Understanding disease“Drops are enough”Poor understanding leads to poor adherenceClear patient education on disease, risks, and expectations
Escalation decisions“Add another drop if pressure rises”Escalation without strategy leads to confusion and side effectsStepwise, purpose-driven escalation or de-escalation
Goal of care“I just need to see clearly”Vision clarity ≠ visual safetyFocus on lifelong preservation of functional vision

FAQs: Glaucoma in Indians

Is glaucoma more common in Indians?

Yes. Indians face a higher risk than many Western populations for two main reasons. First, Indian eyes tend to have a shallower drainage angle, which makes angle-closure glaucoma significantly more common. Second, glaucoma in Indians often develops at a younger age and is detected later, by which time substantial nerve damage has already occurred.


Can Indians get glaucoma even with normal eye pressure?

Yes. Normal tension glaucoma (NTG) occurs when the optic nerve is damaged despite intraocular pressure readings within the normal range. This form is well documented in Indian patients. It is one reason why pressure measurement alone is not enough. A full glaucoma evaluation includes optic nerve imaging and visual field testing.


What are the early signs of glaucoma in Indians?

In most cases, there are no early signs. Glaucoma is called the silent thief of sight because it causes no pain and no blurred vision until the disease is advanced. Peripheral vision goes first, and most people do not notice this until significant damage has occurred. The only reliable way to detect early glaucoma is a comprehensive eye examination.


Who should get screened for glaucoma in India?

Anyone over 40 should have a baseline glaucoma check. Screening is especially important if you have a family history of glaucoma, are short-sighted, have diabetes, use steroid eye drops, or have previously been told your eye pressure is elevated. Earlier screening is recommended if more than one risk factor applies.


How is glaucoma treated in Indian patients?

Treatment depends on the type of glaucoma. Angle-closure glaucoma, which is more common in Indians, often requires laser treatment (laser peripheral iridotomy) in addition to or instead of eye drops. Open-angle glaucoma is typically managed with pressure-lowering drops, laser, or surgery. The right treatment must be matched to the specific type of glaucoma you have, which is why correct diagnosis through gonioscopy and full assessment is essential.

If you have been told you have glaucoma but have not had gonioscopy or a visual field or OCT imaging, a structured second opinion can clarify your diagnosis and treatment plan.

Book a consultation with Dr Shibal Bhartiya:

Marengo Asia Hospitals, Gurugram

Phone: +91 88826 38735

Website: drshibalbhartiya.com

Google Business Profile: maps.app.goo.gl/mcfegmHTuhqV5hSp6

Read the research articles

Read the research articles

This article was written by Dr Shibal Bhartiya, fellowship-trained glaucoma specialist and Mayo Clinic Research Collaborator, Clinical Director at Marengo Asia Hospitals, Gurugram, known for ethical, patient-centred glaucoma care and independent glaucoma second opinions. This article was updated in May 2026.

She has published peer-reviewed research on glaucoma management, examining how treatment decisions should balance medical evidence, patient preferences, and long-term vision outcomes.

As Editor-in-Chief of Clinical and Experimental Vision and Eye Research and Executive Editor of the Journal of Current Glaucoma Practice (Pubmed Indexed, official journal of the International Society of Glaucoma Surgery), Dr Shibal Bhartiya brings editorial and research depth to every clinical decision. Her 200+ publications, including 90+ PubMed-indexed publications and 28 edited textbooks, span glaucoma biology, surgical outcomes, health equity, and emerging diagnostics.

Available on Pubmed and Google Scholar

Dr Shibal Bhartiya
Glaucoma • Second Opinion • Advanced Care

www.drshibalbhartiya.com
 +91 88826 38735

Patient reviews Google Business Profile

Upload your reports for a structured review.

If you are unable to come to Dr Bhartiya’s clinic: Read more about teleconsultation for glaucoma

Steroid Induced Glaucoma

Steroids carry a risk that many patients, and even some prescribing doctors, overlook. They can silently raise the pressure inside your eye. And raised eye pressure, left unchecked, damages the optic nerve and causes glaucoma, says Dr Shibal Bhartiya. Timely monitoring, not waiting for symptoms, is what prevents irreversible optic nerve damage.

Steroids are powerful medicines. Doctors use them to treat inflammation, autoimmune disease, allergies, and dozens of other conditions. But they can trigger a silent rise in eye pressure, often without early symptoms.

This condition is called steroid-induced glaucoma. It is one of the most preventable causes of serious vision loss in India.


Dr Shibal Bhartiya is a fellowship-trained glaucoma specialist and Mayo Clinic Research Collaborator with over 25 years of experience. Her approach focuses on identifying risk before damage is irreversible, simplifying treatment decisions, and protecting vision long-term. Emphasis on early detection, risk assessment, and continuity of care. She is rated 5 stars across 1,500+ patient reviews on Google.


What Are Steroids and Why Do Doctors Use Them?

Steroids, specifically corticosteroids, reduce inflammation in the body. Doctors prescribe them in many forms: eye drops, oral tablets, inhalers, nasal sprays, skin creams, and injections directly into or around the eye.

Common brand names include prednisolone, dexamethasone, betamethasone, triamcinolone, and budesonide. Many are available over the counter in India without a prescription. This is a serious problem.

People often self-medicate with steroid eye drops for redness or allergy, sometimes for months, without any eye pressure monitoring.


How Do Steroids Raise Eye Pressure?

Your eye constantly produces a fluid called aqueous humour. This fluid drains out through a mesh-like structure called the trabecular meshwork. Steroids interfere with this drainage. The fluid builds up. Pressure inside the eye rises.

This process is called a steroid response. It does not happen to everyone. But certain people are far more susceptible. Glaucoma patients, first-degree relatives of glaucoma patients, people with high myopia, and diabetics have a higher risk of becoming steroid responders.

In a steroid responder, eye pressure can rise significantly, sometimes within days of starting treatment. More often, the rise is gradual and goes unnoticed for weeks or months.

The danger is that raised eye pressure causes no pain. No redness. No blurring. You feel nothing until the optic nerve is already damaged.


Which Steroids Carry the Highest Risk?

Eye drops carry the greatest risk. They deliver steroids directly into the eye in concentrated form. Potent drops like prednisolone and dexamethasone raise eye pressure more than weaker formulations like fluorometholone or loteprednol. Duration matters too: the longer the use, the greater the risk.

Periocular injections, injections around the eye used in uveitis and retinal disease, release steroids slowly over weeks to months. Triamcinolone acetonide injections are a particularly common cause of prolonged eye pressure elevation. Once the depot is in place, it cannot be removed easily.

Oral steroids carry a lower but real risk, especially with prolonged use at high doses.

Inhaled steroids for asthma and COPD, and nasal sprays for allergic rhinitis, carry a small but measurable risk, particularly with long-term use.

Skin creams applied around the eyes can absorb through the eyelid skin and raise eye pressure. This is underappreciated and often missed.


Symptoms of Steroid-Induced Glaucoma

In most cases, there are no symptoms. This is what makes steroid-induced glaucoma dangerous.

By the time vision changes become noticeable, significant optic nerve damage has often already occurred. Peripheral vision goes first — and most people do not notice peripheral vision loss until it is severe.

In rare cases, when eye pressure rises very rapidly, patients may experience headache, eye ache, blurring, or haloes around lights. But this is the exception, not the rule.

The only way to detect steroid-induced glaucoma early is to check eye pressure regularly while on any steroid therapy, especially eye drops.


How Is Steroid-Induced Glaucoma Diagnosed?

Diagnosis requires a full glaucoma evaluation. This includes:

Tonometry measures eye pressure. Normal pressure is usually between 10 and 21 mmHg. Steroid responders may reach 30, 40, or even higher.

Gonioscopy examines the drainage angle to confirm the trabecular meshwork is open, as it is in steroid glaucoma, distinguishing it from angle-closure glaucoma.

OCT (Optical Coherence Tomography) scans the optic nerve and the nerve fibre layer to detect structural damage before vision loss is symptomatic.

Visual field testing maps the field of vision to detect functional loss.

Optic disc examination allows direct visualisation of the nerve head for signs of damage and cupping.

Steroid-induced glaucoma looks identical to primary open-angle glaucoma on examination. The distinguishing clue is the history: elevated pressure that developed after starting a steroid, and that improves when the steroid is stopped or changed.


Is Steroid-Induced Glaucoma Reversible?

The short answer: sometimes, if caught early enough.

In many patients, stopping or switching the steroid allows eye pressure to normalise within weeks. If the optic nerve has not been damaged, the condition is fully reversible.

But optic nerve damage is permanent. Glaucoma does not recover. If pressure has been high long enough to injure the nerve, even partially, that damage remains even after the steroid is stopped.

This is why early detection is critical. A short course of steroid eye drops that goes unmonitored can cause permanent vision loss that no treatment can reverse.

Caught early, steroid glaucoma is one of the most manageable forms of glaucoma. That is why monitoring matters.


Treatment Options

Step one is always to reconsider the steroid.

Can the dose be reduced? Can the steroid be stopped? Is there a possibility of using a less potent formulation? For eye drops, switching from prednisolone to fluorometholone or loteprednol often reduces the pressure response significantly.

Sometimes the underlying condition, uveitis, for example, requires continued steroid treatment. In these cases, eye pressure must be managed medically.

Pressure-lowering eye drops are the first line of treatment. The same drops used in primary glaucoma: prostaglandin analogues, beta-blockers, carbonic anhydrase inhibitors, and alpha agonists, are effective in steroid glaucoma.

Laser treatment (SLT) can improve drainage through the trabecular meshwork and reduce dependence on drops.

Surgery: trabeculectomy or a glaucoma drainage device , is reserved for cases where drops and laser do not control pressure adequately. Surgery in steroid glaucoma is generally highly effective.

For patients who have received a periocular steroid injection and cannot have it removed, sustained medical treatment is the mainstay until the depot is absorbed.


The Indian Context: A Hidden Epidemic

India has a particular problem with steroid-induced glaucoma. Steroid eye drops are widely available without prescription. Patients self-treat for red eyes, allergy, and post-operative care, often on the advice of pharmacists or non-specialist practitioners.

Many patients arrive in my clinic having used potent steroid drops every day for six, twelve, or even twenty-four months. Their pressure is grossly elevated. The optic nerves are damaged. Their peripheral vision is affected and will not return.

This is preventable. Every patient using steroid eye drops needs their eye pressure monitored. Every patient on long-term systemic steroids deserves at least an annual eye check. This is not optional.

As a fellowship-trained glaucoma specialist seeing patients from across India, Dr Bhartiya offers structured steroid glaucoma risk assessments for patients on long-term steroid therapy, including those referred by other treating doctors.


When Should You See a Glaucoma Specialist?

See a fellowship-trained glaucoma specialist if:

  • You are using steroid eye drops for more than two weeks
  • You have been prescribed a periocular steroid injection
  • You are on long-term oral steroids and have never had your eye pressure checked
  • You have a family history of glaucoma and are on any steroid therapy
  • You are a known glaucoma patient who requires steroids for any reason
  • Your eye pressure has been noted to be high on a routine eye check
  • If you have been told your eye pressure is high while on steroids, an independent glaucoma second opinion can clarify whether treatment or monitoring is needed.

Do not wait for symptoms. There are none, until it is too late. Bring your steroid prescription and any previous eye pressure readings to your appointment.


Clinical Reality (What’s not always obvious)

  • Steroid-induced glaucoma is often silent in the early stages
  • Vision may remain completely normal on routine testing
  • Pressure rise can happen within weeks in some patients, but months in others
  • Not all steroids are equal — eye drops, skin creams, inhalers, and even nasal sprays can contribute
  • The response is individual — some people are “steroid responders” without knowing it
  • Stopping the steroid does not always reverse the damage completely
  • Damage, once established, follows the same irreversible course as primary glaucoma

What Actually Helps (And What Doesn’t)

What helps:

  • Early identification of steroid use (even non-ocular forms)
  • Baseline and follow-up intraocular pressure monitoring
  • Switching to safer alternatives where possible
  • Timely initiation of anti-glaucoma therapy if needed
  • Long-term monitoring even after stopping steroids

What doesn’t help:

  • Assuming “short-term use is always safe”
  • Ignoring non-eye steroid sources (dermatology creams, inhalers)
  • Relying only on vision clarity as a marker of safety
  • Delaying evaluation because symptoms are absent
  • Repeated steroid prescriptions without pressure monitoring

Remember This

Situation / TriggerWhat Patients Often AssumeClinical RealityWhat Should Be Done
Using steroid eye drops“Doctor prescribed it, so it’s safe”Even prescribed steroids can raise eye pressureMonitor IOP within weeks of starting
Using skin creams near eyes“It’s just topical, not affecting eyes”Periocular absorption can increase eye pressureInform ophthalmologist and monitor
Using inhalers for asthma“It doesn’t reach the eye”Chronic use can contribute to pressure risePeriodic eye pressure checks
Short-term steroid use“Too brief to cause harm”Some individuals respond rapidlyEarly follow-up is essential
No symptoms“If I see well, everything is fine”Glaucoma damage is silent initiallyRegular screening, not symptom-based
Stopping steroids“Problem is solved now”Damage may persist or progressContinued monitoring required
Multiple steroid prescriptions“Different doctors, different issues”Cumulative exposure increases riskCentralised tracking of steroid use

Frequently Asked Questions

Can steroid eye drops cause glaucoma even when used for a short time?

A brief course, less than two weeks, rarely causes a clinically significant pressure rise. But risk increases with duration and potency. Any steroid eye drop use lasting more than two weeks warrants a pressure check.

How long does it take for steroids to raise eye pressure?

In highly susceptible individuals, pressure can rise within days. In most steroid responders, the rise occurs over two to six weeks of use. With depot injections, pressure may continue to rise for months.

Does stopping the steroid cure steroid glaucoma?

It normalises the pressure in most patients, yes. But if the optic nerve has already been damaged, that damage is permanent. Stopping the steroid does not restore lost vision.

Can inhaled steroids for asthma cause glaucoma?

Yes, though the risk is lower than with eye drops. Long-term use of high-dose inhaled corticosteroids has been associated with a modest increase in glaucoma risk, particularly in patients who already have elevated eye pressure.

Can steroid skin creams cause glaucoma?

Yes. Creams applied to the face and eyelid skin can absorb into the eye in meaningful amounts. This is an underrecognised cause of steroid-induced ocular hypertension.

What is a steroid responder?

A steroid responder is someone whose eye pressure rises significantly on steroid therapy. Roughly 5% of the general population are high responders. Glaucoma patients, first-degree relatives of glaucoma patients, high myopes, and diabetics have a much higher rate of response.

Is steroid glaucoma the same as regular glaucoma?

The optic nerve damage is identical. The mechanism of pressure elevation differs: steroids impair drainage through the trabecular meshwork. The treatment approach is similar, but the critical first step is always to reassess and if possible stop or reduce the causative steroid.

Can I still use steroids if I have glaucoma?

Yes, but only under close specialist supervision with frequent pressure monitoring. Never use steroid eye drops without the oversight of an ophthalmologist if you have a diagnosis of glaucoma or a family history of the condition.

I had a steroid injection around my eye six months ago and my pressure is still high. What should I do?

This is a recognised complication of periocular depot steroids. The injection releases slowly over months. Pressure management with drops or laser is usually required until the depot is absorbed. See a glaucoma specialist, this situation requires careful, ongoing monitoring.

What should I do if my pharmacist gives me steroid eye drops for a red eye?

Do not use steroid eye drops without a diagnosis from an ophthalmologist. Red eyes have many causes, viral conjunctivitis, allergic conjunctivitis, dry eye, most of which do not always require steroids and some of which can be worsened by them. Always get a proper diagnosis before using any steroid eye drop.


Book a consultation with Dr Shibal Bhartiya:

Marengo Asia Hospitals, Gurugram

Phone: +91 88826 38735

Website: drshibalbhartiya.com

Google Business Profile: maps.app.goo.gl/mcfegmHTuhqV5hSp6

Read the research articles

This article was written by Dr Shibal Bhartiya, fellowship-trained glaucoma specialist and Mayo Clinic Research Collaborator, Clinical Director at Marengo Asia Hospitals, Gurugram, known for ethical, patient-centred glaucoma care and independent glaucoma second opinions. This article was edited in April 2026.

She has published peer-reviewed research on glaucoma management, examining how treatment decisions should balance medical evidence, patient preferences, and long-term vision outcomes.

As Editor-in-Chief of Clinical and Experimental Vision and Eye Research and Executive Editor of the Journal of Current Glaucoma Practice (Pubmed Indexed, official journal of the International Society of Glaucoma Surgery), Dr Shibal Bhartiya brings editorial and research depth to every clinical decision. Her 200+ publications, including 90+ PubMed-indexed publications and 28 edited textbooks span glaucoma biology, surgical outcomes, health equity, and emerging diagnostics.

Available on Pubmed and Google Scholar

Dr Shibal Bhartiya
Glaucoma • Second Opinion • Advanced Care

www.drshibalbhartiya.com
 +91 88826 38735

Patient reviews Google Business Profile

Upload your reports for a structured review.

If you are unable to come to Dr Bhartiya’s clinic, you can opt for a teleconsult.

How I Think About Glaucoma

I think of glaucoma as a slow, silent risk to lifelong vision rather than just an eye pressure problem. The…